Fibroblast Growth Factor 23/Klotho Axis in Chronic Kidney Disease

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Fibroblast growth factor 23/klotho axis in chronic kidney disease.

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Regulation of fibroblast growth factor-23 in chronic kidney disease.

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Update on Fibroblast Growth Factor 23 in Chronic Kidney Disease

Chronic kidney disease (CKD) is a public health epidemic that affects millions of people worldwide. Presence of CKD predisposes individuals to high risks of end-stage renal disease (ESRD), cardiovascular disease, and premature death. Disordered phosphate homeostasis with elevated circulating levels of fibroblast growth factor 23 (FGF23) is an early and pervasive complication of CKD. CKD is like...

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Fibroblast growth factor 23 is upregulated in the kidney in a chronic kidney disease rat model

The hormone fibroblast growth factor 23 (FGF23) is secreted from bone and is involved in phosphorus (P) metabolism. FGF23 mainly binds the FGF receptor, which interacts with αKlotho in the kidney or parathyroid and regulates Na-dependent phosphate co-transporter type IIa (NaPi-IIa) and type IIc (NaPi-IIc) expression, 1,25-dihydroxyvitamin D3 (1,25(OH)2D3) activity, and parathyroid hormone (PTH)...

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Relationship between Fibroblast Growth Factor-23 and Mineral Metabolism in Chronic Kidney Disease

Fibroblast growth factor- (FGF-)23 is a recently discovered regulator of calcium-phosphate metabolism. FGF-23 appears to decrease in synthesis and accelerated degradation of 1,25(OH)(2)D. Together with its cofactor Klotho, FGF-23 maintains serum phosphate levels within the normal range by increasing renal phosphate excretion. In chronic kidney disease (CKD), FGF-23 levels rise in parallel with ...

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ژورنال

عنوان ژورنال: Nephron Clinical Practice

سال: 2014

ISSN: 1660-2110

DOI: 10.1159/000365787